Bladder pain in BPS/IC may result from an exaggerated response of TRPV1 and enhanced urothelial ATP release induced by chronic adrenergic overactivity

Matos R1, Cordeiro J M2, Igawa Y3, Cruz F4, Charrua A5

Research Type

Basic Science / Translational

Abstract Category

Pelvic Pain Syndromes / Sexual Dysfunction

Abstract 444
Nerves and Bladder Sensations
Scientific Podium Session 29
Thursday 8th October 2015
15:30 - 15:45
510C
Painful Bladder Syndrome/Interstitial Cystitis (IC) Pharmacology Basic Science
1. IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal, 2. Group of animal genetics evolution, CIIMAR, University of Porto, Porto, Portugal, 3. Department of Continence Medicine, University of Tokyo, Tokyo, Japan, 4. Departament of Urology, Hospital São João, Porto, Portugal; Department of Renal, Urologic and Infectious diseases, Faculty of Medicine of University of Porto, Porto, Portugal; IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal, 5. IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal ; Department of Renal, Urologic and Infectious diseases, Faculty of Medicine of University of Porto, Porto, Portugal
Presenter
A

Ana Charrua

Links

Abstract