Changhao Wu
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Podium Session 14 Best Pure and Applied Science 108 Identification of inflammatory factor-mediated signaling pathways for Nox activation in the urothelium by exploration of angiotensin II actions - pathological implications
Podium Session 15 Best Basic Science 231 Identification of the major NADPH oxidase subtype and superoxide production in the micturition centre – pathological implications
Open Discussion Session 30 On Demand Pharmacology 471 Pathological exploration of NADPH oxidase (Nox) derived ROS production from the urothelium in aging and human conditions
Podium Short Oral Session 12 Autonomic Pharmacology 237 NADPH oxidase-driven superoxide generation in bladder urothelium – effect of Nox subtype deletion and the inflammatory mediators
Podium Session 14 Best Basic Science 262 Molecular and functional identification of NADPH oxidase (Nox) in the urothelium: implications for bladder dysfunction and specific ROS controlling targets
Podium Poster Session 25 Urothelial Signalling 229 Dissecting the non-purinergic paracrine mediators in the urothelium-mediated bladder hyperactivity
Podium Poster Session 27 Molecules and Cell Signalling 277 Muscarinic modulation of urothelial ATP release and its paracrine action
Non Discussion Poster Session 17 168 The pathophysiology of human detrusor myogenic overactivity
ICS Member (Expired)
Scientific Committee Member